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In fact, many recreational bodybuilding cycle logs report gaining over 10-15 pounds of muscle from one 12 week cycle of Ibutamoren, and some reports in the literature refer to gaining between 22 and 35 pounds of muscle from 12 weeks of cycle training using Ibutamoren. One study, comparing Ibutamoren to anabolic steroids in 16 participants aged 20-33 years that included 14 heavy resistance trained individuals reported that while Ibutamoren increases muscle mass in 18 to 25 percent of all individuals receiving this prescription, steroids increase muscle mass in around 50 percent of individuals receiving this prescription. As with most drugs that induce growth hormone release, taking Ibutamoren during cycle training can result in adverse effects to the liver. However, these adverse effects may not always be evident on the scale. Some of the following liver-related effects are common with long term use of I butamoren and may not always be noticeable during a given cycle of training: Increased lipid peroxidation. This increase in lipid peroxidation occurs most prominently when the body is exposed to oxidative stress, and this increase in lipid peroxidation results in increased levels of liver damage that may be the cause of the negative effects of the drug. Decreased glutathione. Decreased glutathione is also the body's antioxidant to protect against damage from free radicals and oxidative stress. Due to its antioxidant properties, Ibutamoren can deplete high levels of glutathione in the body leading the body to seek other sources of the antioxidant in order to restore functioning of its protective system. Decreased the enzyme catalase. The liver enzyme catalase breaks down the toxic components of the cell wall and promotes cell repair. Low catalase results in tissue damage and increased blood levels of cholesterol. The liver enzyme catalase breaks down the toxic components of the cell wall and promotes cell repair. Low catalase results in tissue damage and increased blood levels of cholesterol. Decreased sulfate oxidase. Decreased sulfate oxidation also contributes to tissue damage and is involved in the process of the cell's death. As the body's sulfate oxidative system becomes less efficient, the body may be more prone to cell damage and damage can occur even longer periods of time. It is important to note that sulfate oxidation is not something that can occur in a normal body. When a liver malfunctioning due to diabetes, hypertension, or heart disease is exposed to too much sulfate oxidation and its sulfite/sulfate toxicity, a condition known as hyperoxia, it can result in a range of symptoms. Examples of symptoms include extreme thirst, lack of urination Similar articles:
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